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The role of high LET radiation induced persistent oxidative stress and nucleotide excision repair on genomic instability in mammalian cells.
https://repo.qst.go.jp/records/69736
https://repo.qst.go.jp/records/697363f4c9b12-474b-4db1-8ff5-4df1e129ab13
| Item type | 会議発表用資料 / Presentation(1) | |||||
|---|---|---|---|---|---|---|
| 公開日 | 2009-04-13 | |||||
| タイトル | ||||||
| タイトル | The role of high LET radiation induced persistent oxidative stress and nucleotide excision repair on genomic instability in mammalian cells. | |||||
| 言語 | ||||||
| 言語 | eng | |||||
| 資源タイプ | ||||||
| 資源タイプ識別子 | http://purl.org/coar/resource_type/c_c94f | |||||
| 資源タイプ | conference object | |||||
| アクセス権 | ||||||
| アクセス権 | metadata only access | |||||
| アクセス権URI | http://purl.org/coar/access_right/c_14cb | |||||
| 著者 |
Konishi, Teruaki
× Konishi, Teruaki× Hafer, Kurt× Robert, Schiestl× 小西 輝昭× Ｈａｆｅｒ Ｋｕｒｔ× Ｒｏｂｅｒｔ Ｓｃｈｉｅｓｔｌ |
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| 抄録 | ||||||
| 内容記述タイプ | Abstract | |||||
| 内容記述 | N-Acetyl Cysteine (NAC) is a nontoxic radical scavenger and capable radioprotector against low-LET radiation. Last year we measured the NAC protective effect against accelerated neon ion induced ROS and Hprt mutations. Here we report the ability of NAC to protect against neon ion induced clonogenic cell-killing. We observed that NAC protects against x-ray induced cell-killing, we did not observe NAC protection against neon ion induced cell killing. | |||||
| 会議概要（会議名, 開催地, 会期, 主催者等） | ||||||
| 内容記述タイプ | Other | |||||
| 内容記述 | 平成20年度HIMAC共同利用研究成果発表会 | |||||
| 発表年月日 | ||||||
| 日付 | 2009-04-14 | |||||
| 日付タイプ | Issued | |||||
