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Influence of the shielding on the space radiation biological effectiveness
https://repo.qst.go.jp/records/55246
https://repo.qst.go.jp/records/55246b67ee680-a4c1-4bff-99af-5056bbed5b31
Item type | 一般雑誌記事 / Article(1) | |||||
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公開日 | 2004-01-25 | |||||
タイトル | ||||||
タイトル | Influence of the shielding on the space radiation biological effectiveness | |||||
言語 | ||||||
言語 | eng | |||||
資源タイプ | ||||||
資源タイプ識別子 | http://purl.org/coar/resource_type/c_6501 | |||||
資源タイプ | article | |||||
アクセス権 | ||||||
アクセス権 | metadata only access | |||||
アクセス権URI | http://purl.org/coar/access_right/c_14cb | |||||
著者 |
Matsumoto, Hideki
× Matsumoto, Hideki× Hayashi, Saxhiko× Hui, Jin Zhao× Hatashita, Masanori× Kano, Eiichi× Oonishi, Takeo× Furusawa, Yoshiya× 松本 英樹× 大西 武雄× 古澤 佳也 |
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抄録 | ||||||
内容記述タイプ | Abstract | |||||
内容記述 | To investigate whether the bystander effect mediated nitric oxide (NO) radicals excreted from the tumor irradiated with accelerated carbon ion beams modulates growth of the unirradiated tumor at an opposite side in the same mouse. Also, to investigate whether NO generating agents can sensitize the tumoricidal effect by accelerated carbon ion beams. NO at the relatively low concentrations, which excreted from the irradiated donor tumors with accelerated carbon ion beams induced both inhibition and stimulation of tumor regrowth. These findings indicate the importance of an intercellular signal transduction pathway initiated by NO in cellular response to radiation. On the other hand, NO at the relatively high concentrations (0.3 mM) sensitized the tumoricidal effect of X-rays in the p53-independent manner. |
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書誌情報 |
Annual Report of the Research Project with Heavy Ions at NIRS-HIMAC 巻 2002, p. 155-156, 発行日 2003-04 |
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出版者 | ||||||
出版者 | 放医研 |