@misc{oai:repo.qst.go.jp:00086091,
 author = {Kiwamu, Matsuoka and Kosei, Hirata and Naomi, Kokubo and Kenji, Tagai and Hironobu, Endo and Keisuke, Takahata and Hitoshi, Shinoto and Maiko, Ono and Chie, Seki and Kazunori, Kawamura and Zhang, Ming-Rong and Hitoshi, Shimada and Yuhei, Takado and Makoto, Higuchi and Kiwamu, Matsuoka and Kosei, Hirata and Naomi, Kokubo and Kenji, Tagai and Hironobu, Endo and Keisuke, Takahata and Hitoshi, Shinoto and Maiko, Ono and Chie, Seki and Kazunori, Kawamura and Zhang, Ming-Rong and Hitoshi, Shimada and Yuhei, Takado and Makoto, Higuchi},
 month = {May},
 note = {Glutamatergic neurons and cingulate cortices have crucial roles in the cognitive dysfunction of Alzheimer's disease (AD). This study aimed to evaluate regional vulnerabilities of the glutamatergic system in AD at the level of the cingulate gyrus in relation to tau and amyloid-β (Aβ) depositions. Combining MRSI and PET, we found that the glutamatergic system in the posterior cingulate cortex (PCC) is vulnerable to tau deposits but not Aβ from the early stage of AD, and glutamate in the PCC region may be a marker of disease progression in AD., 2022 Joint Annual Meeting ISMRM-ESMRMB},
 title = {Glutamatergic dysfunction associated with tau depositions in Alzheimer’s disease},
 year = {2022}
}