@article{oai:repo.qst.go.jp:00056139,
 author = {小林, 亜利紗 and 及川, 将一 and 古澤, 佳也 and Wang, Jun and アッサワプロンポーン, ナロンチャイ and テングゥ, アーマッド テングゥ アブリザル ファリザル and 小西, 輝昭 and 小林 亜利紗 and 及川 将一 and 古澤 佳也 and アッサワプロンポーン ナロンチャイ and テングゥ アーマッド テングゥ アブリザル ファリザル and 小西 輝昭},
 journal = {平成29年度共用施設（PASTA&SPICE, NASBEE, X/γ線照射装置）成果報告書},
 month = {Dec},
 note = {The aim of radiation cancer therapy as an effective tool is to target the tumor accurately, while limiting the exposure to normal tissue. Although the target is the tumor cells, the exposure effect also occurs in the normal tissue surrounding the tumor area. Therefore, it is essential to investigate the intercellular signaling process between irradiated cells and non-irradiated cells in surrounding areas, known as radiation-induced bystander effect. In previous study, we reported that a signal from the bystander normal cells have modulated the DNA double strand breaks (DSB) repair in irradiated cancer cells exhibiting a rescue effect [1]. However, the question arises now is how radiation-induced bystander effect is facilitated? There are two major pathways that permit cell to cell interaction; Gap-junction and Media Transfer technique. Therefore, in this study we proceed with our previous findings to determine which pathway is involved in rescue effect.},
 pages = {25--27},
 title = {Analysis of radiation-induced bystander response between human normal cells and cancer cells},
 year = {2018}
}