@article{oai:repo.qst.go.jp:00047103,
 author = {Ito, Hiroshi and Shimada, Hitoshi and Shinotoh, Hitoshi and Takano, Harumasa and Sasaki, Takeshi and Nogami, Tsuyoshi and Suzuki, Masayuki and Nagashima, Tomohisa and Takahata, Keisuke and Seki, Chie and Kodaka, Fumitoshi and Eguchi, Yoko and Fujiwara, Hironobu and Kimura, Yasuyuki and Hirano, Shigeki and Ikoma, Yoko and Higuchi, Makoto and Kawamura, Kazunori and Fukumura, Toshimitsu and Lindström, Böö Eva and Farde, Lars and Suhara, Tetsuya and 伊藤 浩 and 島田 斉 and 篠遠 仁 and 高野 晴成 and 佐々木 健至 and 野上 毅 and 鈴木 雅之 and 永嶌 朋久 and 高畑 圭輔 and 関 千江 and 小高 文聰 and 江口 洋子 and 藤原 広臨 and 木村 泰之 and 平野 成樹 and 生駒 洋子 and 樋口 真人 and 河村 和紀 and 福村 利光 and 須原 哲也},
 issue = {6},
 journal = {Journal of nuclear medicine : official publication, Society of Nuclear Medicine},
 month = {Apr},
 note = {Characteristic neuropathologic changes in Alzheimer disease (AD) are amyloid-β deposits and neurofibrillary tangles. Recently, a new radioligand for amyloid senile plaques, (11)C-labeled 5-(6-{[tert-butyl(dimethyl)silyl]oxy}-1,3-benzothiazol-2-yl)pyridin-2-amine ((11)C-AZD2184), was developed, and it was reported to show rapid brain uptake followed by rapid washout. In this study, (11)C-AZD2184 binding in control subjects and AD patients was examined in more detail by compartment model analysis using a metabolite-corrected arterial input function. The accuracy of simplified quantitative methods using a reference brain region was also evaluated.},
 pages = {932--938},
 title = {Quantitative Analysis of Amyloid Deposition in Alzheimer Disease Using PET and the Radiotracer 11C-AZD2184.},
 volume = {55},
 year = {2014}
}