@article{oai:repo.qst.go.jp:00046818,
 author = {Sogawa, Chizuru and Fujiwara, Yasuhiro and Tsukamoto, Satoshi and Ishida, Yuka and Yoshii, Yukie and Furukawa, Takako and Kunieda, Tetsuo and Saga, Tsuneo and 吉井 幸恵 and 古川 高子 and 佐賀 恒夫},
 journal = {Reproductive Biology and Endocrinology},
 month = {Jul},
 note = {C-type natriuretic peptide (CNP) signaling through its receptor natriuretic peptide receptor B (NPR-B) is a key molecule for mammalian reproduction, and known to play important roles in female fertility. However, the function of these peptides in mouse male reproduction remains largely unknown. To determine the role of CNP/NPR-B signaling in male reproduction we investigated phenotype of Npr2-deficient short-limbed-dwarfism (Npr2slw/slw) mice, which have been shown to have gastrointestinal (GI) abnormalities.},
 title = {Mutant phenotype analysis suggests potential roles for C-type natriuretic peptide receptor (NPR-B) in male mouse fertility.},
 volume = {12},
 year = {2014}
}